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and our group reported that macrophage and VMSCs apoptosis is necessary to reduce macrophage burden and stabilize the atherosclerotic plaque within the developing lesion in Apo E −/− mice [67, 68]

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Current laboratory investigations further evaluate intracellular signaling involving cyclic AMP (cAMP) , protein kinase A (PKA) , MAPK , CREB-mediated transcription , JAK/STAT , and additional second messenger systems associated with GHRHR activation